MORTALITY RISK AND SURVIVAL IN THE AFTERMATH OF THE MEDIEVAL BLACK DEATH.


Acid-sensing ion channel-1 contributes to the failure of myelin sheath regeneration following spinal cord injury by transcellular delivery of PGE2

Abstract Background Traumatic injuries to spinal cord lead to severe motor, sensory, and autonomic dysfunction.The accumulation of inhibitory compounds plays a pivotal role in the dea eyewear secondary damage to sparing neural tissue and the failure of axonal regeneration and remyelination.Acid-sensing ion channel-1(ASIC1A) is widely activated foll

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